Tinnitus, the perception of sound without an external source—often described as ringing, buzzing, or hissing—affects millions of people worldwide. For many, it disrupts sleep, concentration, and emotional well-being. While there is no definitive cure, Vagus Nerve Stimulation (VNS) has emerged as a promising treatment option for chronic, treatment-resistant tinnitus.

Tinnitus is thought to result from maladaptive plastic changes in the auditory system, particularly following hearing loss. When auditory input is reduced, the brain may reorganize in a way that increases neural activity in regions linked to sound perception—even in the absence of sound. This abnormal hyperactivity and synchrony may lead to the persistent perception of phantom sounds.

Vagus Nerve Stimulation is a a non-invasive technique that activates the vagus nerve, which plays a central role in regulating various brain functions. Vagus Nerve Stimulation has the potential to modulate brain circuits involved in tinnitus.

Vagus Nerve Stimulation (VNS) promotes the release of neuromodulators like norepinephrine and acetylcholine, which help reshape the brain’s response to sensory input.

In a 2015 clinical study involving 30 patients with chronic tinnitus, transcutaneous Vagus Nerve Stimulation (VNS) led to significant improvements in symptom severity. About 50% of participants reported relief, and both tinnitus loudness and awareness scores improved. No major side effects were observed.

A 2017 study involving 97 patients found that, transcutaneous Vagus Nerve Stimulation (VNS) reduced sympathetic nervous system overactivity, a common feature in tinnitus sufferers. In 75% of cases, heart rate variability improved, suggesting reduced stress and enhanced parasympathetic activity.

Transcutaneous VNS has been shown to be generally safe and well-tolerated.

Vagus Nerve Stimulation represents a promising option for people with chronic, treatment-resistant tinnitus.